Abstract

Exact primary cause for the development of preeclampsia is unknown. However abnormal trophoblastic invasion, inappropriate endothelial cell activation, exaggerated inflammatory response all this may play a key role in the development of preeclampsia. All the pregnancies with inclusion criteria were subjected to Doppler examination after recording the clinical history of the patients, clinical examination and ultrasound after taking informed consent gestational age is calculated by CRL measurement at the time of first trimester scan UA doppler recording were obtained prospectively twice, first at 11 to 14 weeks of Gestation and second at 20 to 24 weeks of gestation. In study group H/O FGR was present in 23 patients (46%), H/O HTN in 27 patients (54%) and previous LSCS in 8 patients (16%). In control group H/O previous LSCS was present in 12 cases (24%). In study group totally 13 HTN cases – 10 had severe preeclampsia, 2 had mild preeclampsia and 1 had gestational HTN. In control group totally 3 had HTN cases 1 had severe preeclampsia, 1 had mild preeclampsia and 1 had gestational HTN.

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