Abstract

Cirrhosis is characterized by fibrosis and the formation of nodules within the liver parenchyma, arising as a consequence of chronic liver injury. This pathological remodeling disrupts the normal lobular architecture of the liver. Cirrhosis is traditionally classified into two stages: compensated and decompensated. The transition from compensated to decompensated cirrhosis is primarily driven by the development of portal hypertension. The onset of decompensated cirrhosis is marked by the emergence of serious complications, including variceal hemorrhage, ascites, spontaneous bacterial peritonitis, hepatic encephalopathy, hepatorenal syndrome, and hepatopulmonary syndrome

Description