Abstract

Myocardial infarction progresses with the duration of coronary occlusion, and the delay in time to reperfusion determines the extent of irreversible necrosis from subendocardial layers towards the epicardium in accordance with the so-called "wave-front phenomenon." Myocardial infarction also progresses with the duration of the coronary occlusion. Myocardial cell necrosis may be prevented by recanalizing the coronary arteries, which can be done either by thrombolytic treatment or primary percutaneous intervention. This can increase the amount of injured but still viable myocardium that can be salvaged from the region that is at risk. In acute myocardial infarction (AMI), magnetic resonance imaging (MRI) can provide a wide range of clinically useful information by detecting not only the location of transmural necrosis, infarct size, and myocardial edema, but also showing in vivo important microvascular pathophysiological processes associated with AMI in the reperfusion era.

Description